Fever is commonly seen in patients with SAH even without infections; its detrimental effects on neurologic outcome, mortality, and vasospasm have been recognized (71,72). Both novel physical (intravascular catheters) (73 ) and conventional pharmacologic means of lowering body temperature are useful; however, many patients after SAH remain refractory to any efforts to achieve normothermia. A multicenter study of intraoperative hypothermia to 33°C that did not follow postoperative fever management failed to show benefit in neurologic outcome (74).

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