Pathogenesis

Typically, a triggering event or combination of events/predisposing factors initiate clotting in a cerebral vein or sinus. When coagulation is activated and thrombosis occurs, the vein, sinus, or frequently both structures become obstructed, with increased resistance to normal venous return to the heart, which leads to a rise in venous and tissue pressure. Most of the consequences of CVT are directly related to some combination of brain edema (tissue congestion), increased ICP, and hemorrhagic infarction.

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