This syndrome is a result of loss of reservoir function when 80% or more of the stomach is removed. It differs from gastric stasis syndrome in that gastric emptying is normal. Symptoms include early satiety, epigastric pain after eating, and vomiting. Some patients develop severe weight loss, malnutrition, and anemia secondary to folate, vitamin B12 or iron deficiency.
Dietary treatment is often successful and consists of increasing the frequency and decreasing the size of the meals, adding supplemental vitamins, iron, and pancreatic enzymes. Surgical treatment aims at increasing gastric capacity by creating a gastric reservoir for the patients with a Hunt-Lawrence pouch (15) or the Tanner Roux-19 pouch (16). Both of these reconstructions carry the risk of stasis and ulceration, and are performed only in patients with severe symptoms that have failed conservative management.
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