Low Testosterone Treatment

Anabolic Running Review

This product is created by Joe LoGalbo who is an American Trainer and professional athletee trainer. Over the time, Joe had been training hundreds of clients per year and got them some incredible results using Anabolic Running. His product consists of four main components including-Anabolic Running Main Product, Stock and Awe strength, Testosterone Hacker Handbook and Indoor Anabolic Running.The main program is the main thing you need in this program. However, there are some bonuses such as Shock and Awe Strength which is a 6-minute exercise sequence which is equally important. You can do this at home any time in order to build strength and dense muscles. The other great thing about the workouts in this program is that they are short and simple to practice. This product is mainly for men who want to save their marriages by increasing testosterone and growth hormones. It is also for those that wants to strip away some fat and get some abs. This product is available in many formats. First of all, the main program is available in readable PDF formats while there are some bonuses available in Video formats to clarify on the results and show you how to go about each of the steps. Read more...

Anabolic Running Review Summary


4.8 stars out of 42 votes

Contents: Ebooks, Videos
Author: Joe LoGalbo
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My Anabolic Running Review Review

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Juicing for your manhood Cure Ed

The creator of this highly helpful program is Olivier Langlois. This man used to be a relatively fat person who had no energy and thought that he is losing a lot in terms of conjugal rights. Olivier realized that he had the Limp Libido Syndrome because of the increase in his testosterone levels in the body. This syndrome was the standing point because he wanted to research and come up with a lasting solution to this problem. The author realized that erectile dysfunctions contributed heavily on the percentage of breakups in marriages. This condition interferes heavily with every aspect of man's life starting from his personal relations to his social life. The man may be living in fear especially if they know their partners are likely to cheat. If the partner starts to cheat, this may cause mental unrest and at the same time stress that would affect people from all aspects of life. With the program, you should fully forget about testosterone injection and other types of medications. The program utilizes natural methods in the treatment of erectile dysfunction. This program is available in PDF formats to make it easy for you to download and print. This will be possible after purchase. Read more...

Juicing for your manhood Cure Ed Summary

Contents: Ebooks
Author: Olivier Langlois
Official Website: juicingforyourmanhood.com
Price: $44.00

Juicing for Your Manhood

Juicing for Your Manhood is a program that employs natural methods in fighting erectile dysfunction that comes about as a result of low testosterone levels. It puts focus in rejuvenating this hormone within the bodies of the affected individuals, transforming them back to the persons they used to be before. It is a creation of Olivier Langlois, a guy who used to be overweight with no energy to satisfy his partners. He was diagnosed with Limp Libido Syndrome due to the reduced level of testosterone hormone within his body. It is this condition that motivated him to carry out extensive research on how he could bring to an end this problem. The ingredients are scientifically and therapeutically verified. Isn't this the most important aspect of a program you may wish to undertake? Definitely yes! The program was designed following a wide-ranging study that is scientifically based. Unlike many programs based on theories, the techniques employed in this guide and been tested and verified practically. The program has no side effects. The techniques used in the guide to upsurge libido are safe, unlike prescriptions. As pointed out at the onset, the program uses natural methods. If you need to regain your manhood, this is a perfect product for you. Read more...

Juicing for Your Manhood Summary

Contents: Ebook
Author: Olivier Langlois
Official Website: juicingforyourmanhood.com
Price: $24.00

31 Day Testosterone Plan

Sick And Tired Of Low Testosterone? This Breakthrough Shortcut Technique Can Help You Unlock Floods Of Natural Free Testosterone In Just 1 Month No Matter What Your Age Or Condition Inside youll learn: The Reason Why Your T Levels are 40% Lower Than Your Grandfathers. The 3 Main Causes of Low-Testosterone (the last one will blow you away). A Unique Liver Flush Technique You Can Use to Remove Excess Estrogen From Your Body. -How Naturally To Increase dopamine, (The libido, pleasure and desire neurotransmitter). -The Man Killing Enzyme That Converts Your Testosterone Into Estrogen and How You Can Get Rid of It, Fast. This is just a Little taste of what youll find inside this e-course. Youll discover super-foods that send your T levels shooting upwards as well as some clear, frank advice on how to steer away from harmful foods that can cause testicular atrophy, man boobs and bedroom performance problems. Read more...

31 Day Testosterone Plan Summary

Contents: Video Course, Ebooks
Author: Mark Wilson
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Are there any illnesses or medical conditions that are associated with osteoporosis

Hypogonadism caused by the illness or condition can be reversible. For example, pregnancy and lactation cause temporary bone loss because calcium is needed by the developing fetus and the infant who is breastfeeding. But pregnancy- and lactation-associated bone loss stops either after delivery of the baby or the weaning of the baby from the breast. Interestingly, this transient bone loss does not have long-term effects and is not changed by taking additional calcium during pregnancy or lactation. Hypogonadism, or low testosterone levels, is a chronic condition that affects men and can interfere with normal bone development. Hypogonadism

Thienopyridinones And Thienopyrimidinones

An initial lead compound 5 was obtained from high throughput screening, and the IC50 of 5 at the rat GnRH receptor is reported to be 3 mM 13 . SAR studies of this series resulted in potent GnRH antagonists such as 6 with IC50 values of 60 nM on the rat GnRH receptor and 2.5 nM at the cloned human GnRH receptor 14-16 . Differences in affinities for GnRH receptors from different species have been reported for this and all other series of nonpeptide GnRH antagonists studied thus far. Functionally, 6 inhibits GnRH-stimulated LH release from rat pituitary cells with an IC50 of 85 nM, and GnRH-stimulated inositol phosphate hydrolysis in CHO cells expressing the human GnRH receptor (IC50 16 nM). A more potent human GnRH antagonist 7 (hGnRH IC50 0.44 nM, PI turnover IC50 1.0 nM) also binds very tightly to the rhesus monkey GnRH receptor (IC50 0.5 nM) and rat GnRH receptor (IC50 4.0 nM), but less at the dog GnRH receptor (IC50 60 nM). This compound suppresses circulating LH and testosterone...

Neuropsychiatry Disorders

Feeding Disorders - Anorexia nervosa is an eating disorder characterized by tremendous weight loss in the pursuit of thinness. There is similar pathophysiology in anorexia nervosa and in depression including the manifestation of hypercortisolism, hypothalamic hypogonadism and anorexia (48,49). Furthermore, the incidence of depression in anorexia nervosa patients is high (50). Like depressed patients, anorexics show a markedly attenuated ACTH response to intravenously administered CRF (48,49) and significantly higher basal CSF levels of CRF than controls (49). When the underweight anorexic subjects are studied after their body weight had been restored to normal, their basal hypercortisolism, increased levels of CRF in the CSF, and diminished ACTH responses to exogenous CRF all return to normal at varying periods during the recovery phase (48,49). CRF can potently inhibit food consumption in rats which further suggests that the hypersecretion of CRF may be responsible for the weight...

Screening For An Endocrine Disorder

A medical history and physical examination should be performed that is directed toward eliciting any symptoms or signs of hyperandrogenism. Screening laboratory tests for hyperandrogenism include a serum DHEAS, total testosterone, free testosterone, and luteinizing hormone follicle-stimulating hormone (LH FSH) ratio. These tests should be obtained apart from the time of ovulation in order to avoid the surge of hormones associated with ovulation. From a practical standpoint, it may be easiest to suggest that women have these tests performed either just prior to or during the menstrual period. It is important to note that if a patient is on oral contraceptives at the time of hormonal testing, an underlying hyperandrogen-emia maybe masked. This does not occur with antiandrogens such as cyproterone or spironolactone. Therefore, it is best that patients discontinue oral contraceptives four to six weeks prior to the endocrine evaluation. An ovarian source of excess androgens can be...

Effect of Hyperinsulinemia on Hyperandrogenism in PCOS

In vivo data also suggest that hyperinsulinemia is associated with an increased level of circulating testosterone in PCOS. When pancreatic insulin secretion was suppressed in women with PCOS by diazoxide, both serum total and free testosterone levels decreased (8). However, diazoxide's suppression of insulin release in healthy normal women did not result in changes in serum testosterone levels (9). Hence, hyperinsulinemia seems to play a role in hyperandrogenemia in PCOS, and women with PCOS may be more susceptible than normal women to stimulation of ovarian androgen production by insulin.

Testosterone Role In Male Sensitivity To Ischemia

Despite the fact that male sex is a well-acknowledged risk factor for human stroke, most research aimed at understanding gender differences in stroke has focused exclusively on female sex steroids. Available epidemiologic studies suggest that testosterone, the major mammalian androgen, has a neutral or favorable effect on cardiovascular disease (43). This possibility is likely related, in part, to beneficial effects on vascular endothelial function and the vasodila-tory properties of testosterone at physiologic concentrations. In the clinical setting, testosterone declines in men after stroke, presumably as a stress response (44). However, testosterone has recently emerged as another sex steroid that has the potential to alter ischemic cell death. For example, surgical castration and subsequent low testosterone decrease histologic damage after focal cerebral ischemia in the young adult male rat (45,46) and negatively affect outcome from experimental spinal cord injury (47). When...

Metformin Weight Loss and PCOS

It is likely that weight reduction plays the most significant role in restoration of ovulation in obese women with PCOS and that this may be the primary mechanism for the effects noted with metformin, independent of its insulin-sensitizing effects (14). More recently, drugs that target weight loss have been compared with metformin therapy in women with PCOS. Treatment with orlistat for 3 months led to a 4.7 reduction in body weight, a more significant loss than was observed in metformin-treated women (1.0 ). Serum testosterone levels declined significantly in both groups with no change in levels of SHBG. This agent has potential as an adjunct to the treatment of PCOS (15). The effects of metformin (1700 mg day) vs acarbose (300 mg day) on insulin resistance, hormone profiles, and ovulation rates in patients with clomiphene citrate-resistant PCOS have recently been assessed (16). The ratio of LH to follicle-stimulating hormone (FSH) and total testosterone concentrations decreased and...

Causes and Symptoms of Smell Disorders

Currently we distinguish between congenital anosmia occurring as an isolated defect or occurring within the context of a syndrome 191 . Isolated congenital anosmia seems to occur more often than previously believed. Apart from the typical patient history of no odor memories, only MR imaging leads to a more definitive diagnosis 192, 193 . In the frontal imaging planes just tangential to the eye bulbs, hypoplastic or aplastic olfactory bulbs can be visualized. This plane also allows an evaluation of the olfactory sulcus which is flattened if the olfactory bulb is absent or aplastic. This is a useful indicator of congenital anosmia, especially since the bulb is not always easy to identify. Among cases of congenital anosmia as part of a syndrome, the Kallmann-Syndrom 194 is the disorder in which it is most frequently encountered. This is an anosmia associated with hypogonado-tropic hypogonadism clinically characterized by infertility and anosmia, where infertility can be reversed by...

Anemia due to CKD in the Elderly

CKD has been reported to be the principal cause of anemia in more than 8 of older subjects, and a contributing cause in others (27). The pathophysiologic basis for anemia related to CKD in older individuals is likely similar to that of younger subjects, complicated perhaps by a greater impact of underlying inflammatory processes, and in men, an age-related decline in testosterone levels (28). Although information is conflicting, data from the Baltimore Longitudinal Study on Aging revealed a rise in serum erythropoietin level over time in that sample of older adults, regardless of the presence or absence of anemia (29, 30). The increase in erythropoietin levels was less marked in subjects with hypertension or diabetes mellitus, perhaps reflecting some underlying CKD, either age-related or due to these other conditions. While some studies suggest that the erythropoietin production in response to anemia and erythropoietin responsiveness is blunted in healthy elderly subjects, others have...

Sexual Desire and Aging

Hormone levels play a role in men's sexual expression, and changes in hormone levels may account for some of the age-related changes in sexuality. Testosterone is the key hormone that regulates sexual response in men and has some effect in women. Testosterone is produced by the adrenal glands, testicles, and ovaries. Women produce less than a tenth of the amount produced by men. Testosterone levels in men are highest in the morning right after waking and decrease throughout the day, so blood tests for testosterone are taken before 9 00 am for accuracy. After a rise during adolescence, testosterone production declines throughout life in men. Much less is known about age-related production of testosterone in women.

Gonadotropin Ovulation Induction

In general, ovulation induction with gonadotropins in clomiphene-resistant PCOS patients is less successful than in patients with hypogonadotrophic hypogonadism (103). However, women with PCOS are more sensitive to gonadotropin stimulation compared with spontaneously cycling women. This increased sensitivity appears to result from a larger pool of small antral follicles available for recruitment, rather than on differences in the FSH threshold level (104). Women with PCOS receiving gonadotropins for ovulation induction are particularly prone to a higher risk of overstimulation, multiple pregnancy, and OHSS rates (105).

Leptin and Bone Metabolism

Mice Leptin Level

In contrast to the evidence for a positive peripheral role of leptin on bone metabolism, ob ob, and leptin receptor-deficient (db db) mice presented a phenotype characterized by increased bone mass with an increased rate of bone formation despite hypogonadism and hypercortisolism 129 . In this study, there was no expression of leptin receptors on osteoblasts, whereas the intracerebroventricular infusion of leptin decreased bone formation rate in ob ob mice as well as in wild-type animals 129 . Mice knock-in of LacZ in the leptin locus with increased circulating leptin level had a dramatically reduced bone mass 130 . In this experimental model, leptin synthesis was not detected in the CNS. As such, circulating leptin must therefore account for the observed antiosteogenic eVect with similar amounts of leptin needed to aVect both body weight and bone mass 130 .


Recent evidence has shown that adipose tissue is an active participant in maintaining energy and glucose homeostasis and plays crucial roles in controlling neuroendocrine, autonomic, and immune functions. Leptin is a hormone secreted by adipose tissue. Deficiency of leptin or its receptor results in hyperphagia, morbid obesity, insulin resistance, hyperlipidemia, hypothalamic hypogonadism, and immunosuppression. These abnormalities are reversed by leptin treatment in patients with congenital leptin deficiency or lipodystrophy. In contrast, diet-induced obesity is associated with elevated leptin levels and blunted response to leptin. Leptin resistance has been ascribed to the reduced entry of leptin into the brain or impairment of leptin signal transduction. This chapter focuses on the current understanding of leptin's actions, with particular emphasis on transport across the blood-brain barrier, signaling via JAK-STAT and PI3-kinase, neuropeptide targets, and electrophysiological...

Stress Management

Muscles tighten as stress starts, often causing intense headaches, backaches, and gastrointestinal problems. Stress also can cause testosterone levels to decrease and blood vessels in the penis to constrict, often resulting in erection problems. The rush of hormones caused by a stressful situation can bring on an asthma attack in a person with a history of asthma. Stress also draws the blood supply away from the abdominal area and encourages overproduction of acids in the digestive system, often leading to indigestion and other gastrointestinal problems. Other problems related to stress include insomnia and irritability.


Compound 22 is much more hydrophilic than the highly lipophilic AG-045572. This compound has Ki values of 40 nM at the human GnRH receptor and 520 nM at the rat receptor. The KB value of this compound for inhibition of GnRH-stimulated inositol phosphate hydrolysis is 30 nM. Compound 23, which is apparently designed to address the possible poor cell permeability of guanidine 22, has an aminopyrimidine structure 33 . This compound has high binding affinity for both rat and human GnRH receptors (hGnRH Ki 9.3 nM, rGnRH Ki 4.7 nM). In cells expressing recombinant rat GnRH receptors, 23 is a competitive inhibitor of GnRH-stimulated extracellular acidification with a pA2 value of 8.3, which is similar to its pKi (8.3) at the rat pituitary receptor. In castrated male rats, 23 dose-dependently suppresses circulating LH levels when given intravenously (5-20 mg kg). When administrated in intact male rats, 23 significantly lowers circulating testosterone levels for up to...

Perimenopausal Stage

Burger and associates (35) also conducted longitudinal studies of women through the menopausal transition. As found by others, they noted little, if any, change in total testosterone levels leading up to, during, and for several years after menopause. However, reductions in SHBG levels were observed in concert with the reduction in estradiol observed leading up to the menopause and thereafter. The net effect of declining SHBG levels in the face of unwavering levels of testosterone is to increase the free testosterone fraction by 80 over the interval beginning 4 years prior to menopause and ending 2 years after the menopause. If free testosterone levels are actually increased during the perimenopausal period, this could have physiological impacts on androgen-dependent systems, such as libido, in these women. Although the utilization of hormone replacement therapy (estrogen with or without a progestin) in postmenopausal women is in a period of flux because of concerns relating to...

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